Investigations into the aetiopathogenesis of orofacial granulomatosis using multiple omics technologies reveals a potential role for B cells
Download from source ↗Dataset overview
Participants
75
Samples
None
Reuse readiness
3.7/10
evidence-backed score
Investigations into the aetiopathogenesis of orofacial granulomatosis using multiple omics technologies reveal a potential role for B cells.
Study facts
- Organism
- Homo sapiens
- Platform
- —
- Age group
- —
- Disease groups
- Non-IBD controls
- Anatomical sites
- —
Data availability
Specific data assets have not been resolved from the source yet — see the source repository below for the full file listing.
Strengths & limitations for reuse
Strengths
- Participant counts are documented
Limitations
- Not documented: raw reads are advertised
- Not documented: feature/otu tables are advertised
- Not documented: taxonomic tables are advertised
Extraction evidence & provenance
Each extracted field is shown with the source excerpt and location used to resolve it.
Assay
| Field | Value | Evidence |
|---|---|---|
assay.sequencing_type |
amplicon_16s |
The oral microbiome of participants was determined using 16S rRNA sequencing. Section |
Cohort
| Field | Value | Evidence |
|---|---|---|
cohort.non_ibd_controls |
43 |
Thirty-two participants with OFG, with and without CD, and forty-three healthy controls were recruited. Section |
cohort.total_participants |
75 computed |
Thirty-two participants with OFG, with and without CD, and forty-three healthy controls were recruited. Section |
Specimens
| Field | Value | Evidence |
|---|---|---|
specimens.body_site |
oral |
The oral microbiome of participants was determined using 16S rRNA sequencing. Section |